Mutant α-synuclein propagates via the lymphatic system of the brain in the monomeric state

PQBP5/NOL10 maintains and anchors the nucleolus under physiological and osmotic stress conditions (2023)

Tau activates microglia via the PQBP1-cGAS-STING pathway to promote brain inflammation (2021)

HMGB1 signaling phosphorylates Ku70 and impairs DNA damage repair in Alzheimer’s disease pathology (2021)

Prediction and verification of the AD-FTLD common pathomechanism based on dynamic molecular network analysis (2021)

Hepta-Histidine Inhibits Tau Aggregation (2021)

DNA damage in embryonic neural stem cell determines FTLDs’ fate via early-stage neuronal necrosis (2021)

YAP-dependent necrosis occurs in early stages of Alzheimer's disease and regulates mouse model pathology (2020)

The intellectual disability gene PQBP1 rescues Alzheimer’s disease pathology.

Ser46-Phosphorylated MARCKS Is a Marker of Neurite Degeneration at the Pre-Aggregation Stage in PD/DLB Pathology

Insights into familial middle-age dementia suggest new avenues for treatment (2018)

Preclinical pathology during development affects the post-onset adulthood symptoms in spinocerebellar ataxia type 1 (2017)

Starvation induced autophagy enhances accumulation of amyloid-beta (Aβ) in neurons (2015)

Molecular basis for the earlist synapse pathology in preclinical Alzheimer's disease brain (2014)

A molecular pathomechanism of microcephaly by mutations of PQBP1 gene (2014)

VCP, a causative gene of FTLD controls the common pathology of polyglutamine diseases (2013)

Intellectual disability gene PQBP1 affects lifespan (2012)

A common mechanism underlying two polyglutamine diseases (~2011)